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By A. Barry Kay, Jean Bousquet, Patrick G. Holt, Allen P. Kaplan

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This quantity publication is a phenomenal reference resource on all elements of allergic reaction and allergic illnesses. masking almost each allergic , from the immunological and molecular foundation of the allergic reaction to destiny traits in allergic affliction prevention, this new foreign editorial workforce (A.B.Kay, Jean Bousquet, Pat Holt and Allen Kaplan) have thoroughly revised and up-to-date the textual content, from either a systematic and medical viewpoint. References will remain additional to the textual content till it is going to press making this the main updated publication to be had within the field.

This moment variation involves greater than 1,800 pages contained inside of ninety eight chapters.

The fee contains a totally searchable spouse CD ROM with the entire textual content and over three hundred pictures from the ebook in complete colour.

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Example text

T. 1 (Cont’d ) 1915 Warfield T. Longcope Experimental lesions in organs from animals repeatedly injected with foreign proteins 1916 Robert A. A. Ramirez Horse dander asthma following blood transfusion 1921 Carl Prausnitz Heinz Küstner Passive transfer of immediate skin reactivity of fish allergen by intradermal injection of serum from allergic patient (“reagins”) 1921 Arent de Besche Passive transfer of serum from horse asthmatics sensitized to horse proteins by injection of diphtheria vaccine 1922 Fernand Widal Pierre Abrami J.

21) had noted similar crystals from a leukemic spleen, hence Charcot–Leyden crystals. qxd 4/1/08 20:04 Page 12 PART 1 Immunology of the Allergic Response Fig. 20 Henry Hyde Salter (1823–71). Description of the various causes of asthma and of cells (now known as eosinophils) in sputum. ) Fig. 22 Francis M. Rackemann (1887–1973). ) Many clinicians appreciated that asthma had an allergic component but it was Francis Rackemann (1887–1973) (Fig. 22) who introduced the term “intrinsic asthma” to describe patients who gave no history of “allergy” and who were skin test negative to common allergens.

There is growing interest in the possible role of Th17 cells in allergic disease although their role remains ill-defined in humans. As discussed in Chapter 3, they are distinct from Th1 and Th2 cells are involved in the initiation of a predominantly neutrophil-rich inflammatory response (Romagnani 2006). Control of allergic inflammation by natural and inducible T regulatory cells is discussed in Chapter 4. Some hypersensitivity reactions do not fall neatly into the type 1–IV classification. For example, activation of the plasma cascade via factor XII, prekallikrein, and high-molecularweight kininogen leads to bradykinin formation, the critical mediator of hereditary angioedema (Fields et al.

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