Download BRS Pathology by Arthur S. Schneider MD, Philip A. Szanto MD PDF
By Arthur S. Schneider MD, Philip A. Szanto MD
You also will discover:
- Full-color layout, illustrations, and tables summarize info for handy review
- Over 450 USMLE-style questions, solutions, and rationales either electronically and in print to augment your pathology overview
- Key subject icons support concentration your examine
- Correlation containers emphasize connections among pathology and medical medication
- A loose spouse site with entry to the publication, photo financial institution, and an interactive query financial institution that includes the entire questions from the e-book for attractive, powerful attempt preparation!
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Extra info for BRS Pathology
B. Notable deficiencies include C2, C3, and C5. Chronic granulomatous disease of childhood (1) This disease is most commonly an X-linked disorder characterized by the deficient activity of one of the enzymes involved in NADPH oxidase activity and the oxidative burst. Autosomal recessive variants also occur. (2) The disease is marked by phagocytic cells that ingest but do not kill certain microorganisms. (3) Catalase-positive organisms are ingested but not killed. , Staphylococcus aureus) can destroy H2O2 generated by bacterial metabolism.
3) It is usually caused by bacterial infections, often by staphylococci. Ulcer (1) This is the loss of surface epithelium. , peptic ulcer and ulcers of the skin). Fistula. This is an abnormal communication between two organs or between an organ and a surface. Scar. This is the final result of tissue destruction, with resultant distortion of structure and, in some cases, altered function. Conversion to chronic inflammation a. This change is marked by the replacement of neutrophils and monocytes with lymphocytes, plasma cells, and macrophages.
A 26-year-old African-American woman has bilateral hilar adenopathy, and radiography reveals multiple reticular densities in both lung fields. A bronchoscopic biopsy reveals granulomatous inflammation with multiple giant cells of the Langhans type and no evidence of caseous necrosis. Which of the following is the most likely diagnosis? (A) (B) (C) (D) (E) Aspergillosis Coccidioidomycosis Histoplasmosis Sarcoidosis Tuberculosis 8. In a laboratory exercise for medical students, an unknown compound is studied.